That is, stimulation of CD74 with exogenous MIF may well lead to an extreme inflammatory condition, as MIF is able to stimulating CXCR2 and CXCR4 receptors In combination with CD74. CXCR2 and CXCR4 receptors when activated promote inflow of neutrophils and lymphocytes, respectively (63, 64). In contrast, activation of the pro-survival kinase AMPK
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